Home Jurnale medicale Journal of the American Society of Nephrology current issue
Jurnale medicale
Journal of the American Society of Nephrology&n...
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Jurnale medicale Journal of the American Society of Nephrology current issue

30 Ianuarie 2015 18:01

Although APOL1 gene variants are associated with nephropathy in African Americans, little is known about APOL1 protein synthesis, uptake, and localization in kidney cells. To address these questions, we examined APOL1 protein and mRNA localization in human kidney and human kidney-derived cell lines.

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01
30 Ianuarie 2015 18:01

Hyperglycemia results in increased oxygen consumption and decreased oxygen tension in the kidney. We tested the hypothesis that activation of hypoxia-inducible factors (HIFs) protects against diabetes-induced alterations in oxygen metabolism and kidney function. Experimental groups consisted of cont

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01
30 Ianuarie 2015 18:01
30 Ianuarie 2015 18:01
30 Ianuarie 2015 18:01

African Americans are at increased risk for cardiovascular and metabolic diseases, including obesity, high BP, diabetes, CKD, myocardial infarction, and stroke. Here we summarize the current risks and provide an overview of the underlying risk factors that may account for these associations. By revi

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Loss of podocytes underlies progression of CKD. Detachment of podocytes from the glomerular basement membrane (GBM) rather than apoptosis or necrosis seems to be the major mechanism of podocyte loss. Such detachment of viable podocytes may be caused by increased mechanical distending and shear force

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01
30 Ianuarie 2015 18:01

Anion exchanger-1 (AE1) mediates chloride-bicarbonate exchange across the plasma membranes of erythrocytes and, via a slightly shorter transcript, kidney epithelial cells. On an omnivorous human diet, kidney AE1 is mainly active basolaterally in α-intercalated cells of the collecting duct, where it

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Indoxyl sulfate and p-cresol sulfate have been suggested to induce kidney tissue remodeling. This study aimed to clarify the molecular mechanisms underlying this tissue remodeling using cultured human proximal renal tubular cells and half-nephrectomized mice treated with indoxyl sulfate or p-cresol

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

A noninvasive means to predict the onset and recurrence of lupus nephritis (LN) before overt renal injury is needed to optimize and individualize treatment. Colony-stimulating factor-1 (CSF-1) is expressed by kidney tubules at the onset of LN, increases with disease progression, and spills into the

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

A variety of genetic backgrounds cause the loss of function of thiazide-sensitive sodium chloride cotransporter, encoded by SLC12A3, responsible for the phenotypes in Gitelman syndrome. Recently, the phenomenon of exon skipping, in which exonic mutations result in abnormal splicing, has been associa

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Conventional markers of macrophages (Mfes) and dendritic cells (DCs) lack specificity and often overlap, leading to confusion and controversy regarding the precise function of these cells in kidney and other diseases. This study aimed to identify the phenotype and function of renal mononuclear phago

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Podocytes are critically involved in the maintenance of the glomerular filtration barrier and are key targets of injury in many glomerular diseases. Chronic injury leads to progressive loss of podocytes, glomerulosclerosis, and renal failure. Thus, it is essential to maintain podocyte survival and a

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Proteinase 3 (PR3) and myeloperoxidase (MPO) are two major autoantigens in patients with vasculitis with ANCA. The genes encoding these autoantigens are abnormally expressed in peripheral granulocytes of patients with active ANCA-associated vasculitis. This study provides evidence that this transcri

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Phospholipase A2 receptor 1 (PLA2R) is a target autoantigen in 70% of patients with idiopathic membranous nephropathy. We describe the location of a major epitope in the N-terminal cysteine-rich ricin domain of PLA2R that is recognized by 90% of human anti-PLA2R autoantibodies. The epitope was sensi

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Membranous nephropathy (MN) is a common cause of nephrotic syndrome in adults. Recent clinical studies established that >70% of patients with idiopathic (also called primary) MN (IMN) possess circulating autoantibodies targeting the M-type phospholipase A2 receptor-1 (PLA2R) on the surface of glomer

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

AKI is associated with increased morbidity, mortality, and cost of care, and therapeutic options remain limited. Reactive oxygen species are critical for the genesis of ischemic AKI. Stanniocalcin-1 (STC1) suppresses superoxide generation through induction of uncoupling proteins (UCPs), and transgen

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Whether inclusion of the coronary artery calcium score improves cardiovascular risk prediction in individuals with CKD, a population with unique calcium-phosphate homeostasis, is unknown. Among 6553 participants ages 45–84 years without prior cardiovascular disease in the Multi-Ethnic Study of Ather

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

ANCA-activated phagocytes cause vasculitis and necrotizing crescentic GN (NCGN). ANCA-induced phagocyte NADPH oxidase (Phox) may contribute by generating tissue-damaging reactive oxygen species. We tested an alternative hypothesis, in which Phox restrains inflammation by downregulating caspase-1, th

mai mult
Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Aldosterone-independent mechanisms may contribute to K+ homeostasis. We studied aldosterone synthase knockout (AS–/–) mice to define renal control mechanisms of K+ homeostasis in complete aldosterone deficiency. AS–/– mice were normokalemic and tolerated a physiologic dietary K+ load (2% K+, 2 days)

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Patients with Gitelman syndrome (GS), an inherited salt-losing tubulopathy, are usually treated with potassium-sparing diuretics or nonsteroidal anti-inflammatory drugs and oral potassium and magnesium supplementations. However, evidence supporting these treatment options is limited to case series s

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Arteriovenous fistula (AVF) is the preferred vascular access for hemodialysis (HD). However, many AVFs fail before starting dialysis. To assess the optimal time for AVF placement in the elderly, we linked data from the US Renal Data System with Medicare claims data to identify 17,511 patients≥67 yea

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Antibody-mediated rejection (AMR) is a major cause of kidney graft loss, yet assessment of individual risk at diagnosis is impeded by the lack of a reliable prognosis assay. Here, we tested whether the capacity of anti-HLA antibodies to bind complement components allows accurate risk stratification

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

Patients on dialysis require phosphorus binders to prevent hyperphosphatemia and are iron deficient. We studied ferric citrate as a phosphorus binder and iron source. In this sequential, randomized trial, 441 subjects on dialysis were randomized to ferric citrate or active control in a 52-week activ

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

An association between atherosclerosis and osteoporosis has been reported in several studies. This association could result from local intraosseous atherosclerosis and ischemia, which is shown by limb osteoporosis in patients with peripheral artery disease (PAD), but also could result from bidirecti

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Journal of the American Society of Nephrology current issue
30 Ianuarie 2015 18:01

High concentrations of HDL cholesterol are considered to indicate efficient reverse cholesterol transport and to protect from atherosclerosis. However, HDL has been suggested to be dysfunctional in ESRD. Hence, our main objective was to investigate the effect of HDL cholesterol on outcomes in mainte

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Journal of the American Society of Nephrology current issue
31 Decembrie 2014 18:01
31 Decembrie 2014 18:01

In children, sporadic nephrotic syndrome can be related to a genetic cause, but to what extent genetic alterations associate with resistance to immunosuppression is unknown. In this study, we designed a custom array for next-generation sequencing analysis of 19 target genes, reported as possible cau

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Journal of the American Society of Nephrology current issue
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